In this case, our patient was treated successfully by endovascular techniques, thereby avoiding major surgery. Furthermore he tolerated the procedure well

and made an uneventful recovery. None of the authors has declared any conflict of interest within the last three years which may arise from being named as an author on the manuscript. “
“The term ‘agenesis’ is taken to mean Partial or almost complete absence of growth in the lung.1The rarity of this condition is AUY-922 order evident by the infrequent reporting of such cases in literature with prevalence of 34 per million live births. Till 1970 only 220 cases were reported world wide. Needless to say, bilateral agenesis is incompatible with life. Unilateral agenesis of the lung is much less rare and may present with varying degrees of severity. They are often wrongly diagnosed for more common conditions of unilateral volume loss and it is even more challenging if it comes to notice in adult life.Here we report a case of young man presenting with left pulmonary agenesis. A 24 year old male presented with insidious onset, progressive shortness of breath since childhood and frequent episodes of cough with mucopurulent sputum, often one cupful per day, yellowish in colour. There were no

Tenofovir solubility dmso history of orthopnea, palpitation, wheezing, chest pain, coughing out of blood,anorexia and weight loss.He had no past history suggestive of pulmonary tuberculosis. His perinatal history

was insignificant and no history of similar complaints in any of his siblings. On examination, he was an average built male, malnourished, preferring Adenosine triphosphate left lateral decubitus. Pallor, icterus, clubbing, engorged neck veins and lymphadenopathy were absent. Central cyanosis and pitting pedal oedema were present.On inspection of chest,accessory muscles were working,drooping of shoulder seen in left side and scoliosis with convexity to right noticed.Intercostal suction was seen.On palpation,movement diminished in left side with rib crowding,trachea deviated to left and apex beat placed at left 6th intercostal space in mid axillary line.Expansion of chest was 3 cm and vocal fremitus diminished throughout the left side.On percussion,left side had impaired note 7th ICS downward along MAL and scapular line,resonant in rest of the areas.On auscultation,bilateral vesicular breath sound heard with reduced vocal resonance on left side,bilateral coarse crepitations heard in inter and infrascapular area and right axillary region.Liver was palpable by 2 cm.S2 was loud,other systems were within normal limits. Chest radiograph showed homogenous opacity in the left lower zone, obliterating the left costophrenic angle with gross shifting of the mediastinum to the left and scoliosis with convexity to the right and reticulonodular shadows in the right lower zone(Fig. 1).ECG showed tall peaked P waves in lead 2.