These nanocomposites were prepared by one-step in situ deposition oxidative polymerization of pyrrole hydrochloride using ferric chloride (FeCl3) as an oxidant in the presence of find more ultra fine grade powder of anatase TiO2 nanoparticles cooled in an ice bath. The obtained nanocomposites were characterized by Fourier-transform infrared (FTIR), thermogravimetric analysis (TGA), X-ray diffraction (XRD), and scanning

electron microscope (SEM) techniques. The obtained results showed that TiO2 nanoparticles have been encapsulated by PPy with a strong effect on the morphology of PPy/TiO2 nanocomposites. Also, the synthesized PPy/TiO2 nanocomposites had higher thermal stability than that of pure PPy. The investigation of electrical conductivity of nanocomposites by four-point probe instrument showed that the conductivity of nanocomposite at low TiO2 content is much higher than of neat PPy, while with the increasing contents of TiO2, the conductivity decreases. (C) 2011 Wiley Periodicals, Inc. J Appl Polym Sci 123: 1922-1927, 2012″
“Background. Pre-exposure to hyperoxic gas (>= 95%) has been shown to protect the heart and central nervous system from ischemia-reperfusion injury. In the present study, we investigated whether oxygen pretreatment induces

delayed renal protection OICR-9429 in rats. The possible role of some renal antioxidant agents was also investigated. Materials and methods. Adult male Wistar rats were kept in a hyperoxic (HO) (>= 95% O(2)) environment for 0.5 h, 1 h, 2 h, 3 h, 6 h, and 2 h/day for three consecutive days and 4 h/day for six consecutive days, and control group (IR) animals were kept in the cage with no HO, one day before subjecting their kidney to 40 minutes of ischemia and 24h of reperfusion. Renal function was assessed by comparing plasma www.sellecn.cn/products/cl-amidine.html creatinine

(Cr), blood urea nitrogen ( BUN), creatinine clearance (CLCr), and fractional excretion of sodium (FENa%). Histopathological injury score was also determined according to the Jablonski method. To examine the antioxidant system induction by hyperoxia, we measured renal catalase and superoxide dismutase activity, and renal glutathione and malondialdehyde content. Results. Our data demonstrated that only in 4 h/day HO for six consecutive days, the renal function tests (Cr, CLCr, BUN, and FENa%) and Jablonski histological injury were better than control group (p < 0.05). The beneficial effect of oxygen pretreatment in this group was associated with increased renal catalase activity compared with those obtained from control group ( p < 0.05). Conclusion. The present study demonstrates that repeated exposure to hyperoxic (>= 95% O(2)) environment can reduce subsequent rat’s renal ischemia-reperfusion damage.